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Insulin Resistance (E88.81): Diagnostics and Correction Protocol

Insulin resistance is a condition in which the body's cells stop responding effectively to insulin, forcing the pancreas to produce more of it to keep blood glucose within normal range. It is the core mechanism behind type 2 diabetes, metabolic syndrome, and PCOS, and is assessed using the HOMA-IR index and fasting insulin levels. This page brings together the three stages of managing the condition: metabolic diagnostics, the correction protocol, and how the online consultation works.

The 50+ biomarker panel includes the HOMA-IR index, fasting insulin and glucose, HbA1c, a lipid profile (triglycerides, HDL), liver enzymes as a marker of hepatic fat accumulation, CRP as a marker of chronic inflammation, and, where indicated, sex hormones (relevant in PCOS), a thyroid panel, ferritin, and vitamin D.

The standard workup most patients receive by default — fasting glucose, sometimes HbA1c — misses early-stage insulin resistance: glucose can stay within normal range while insulin is already compensating by running high. Without HOMA-IR and fasting insulin, this stage stays invisible until glucose metabolism is already disrupted, and without the wider panel, the contributing drivers — inflammation, hepatic fat, hormonal imbalance — go unseen.

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